There are several programs that must be resident on your computer to use GMOMMWEB. All of the programs are free downloads:, and Shockwave Player full version 10.x (or later) from Adobe (Macromedia): or Some movies open using Adobe's.
Like QuickTime Player (7.x), Flash Player (9.x) is a free download. The program requires Pentium PCs running Windows 2000 or XP or Mac Computers (G3, G4, G5) running OSx (version 10.3.9 or later)or Intel Macs running OS 10.4 or Windows XP. 500 MHz or faster processor with at least 256 MB RAM required. The program opens in a browser window: Microsoft Internet Explorer (version 5.x or later) provides consistent results on both PCs and Macs. Safari or IE may be used on Macs. See GMOMMWEB Instructions pdf for minimal & recommended computer specifications. Print a copy of this Adobe pdf file.
Printing is now available for many of the pages in GMOMMWEB. Get word for mac. A PrintOmatic 'Xtra' is required.
You may be prompted to download the FREE Xtra from Electronic Ink if it is not in the Shockwave 10 Xtras Folder. NOTE: If you run Apple's OS 10 on an Intel Mac please see Smalldog gives you THREE WAYS to access to information in the GMOMMWEB program: 1. Open the program and choose a topic to study from the list of all topic areas. Click this link: 2.
Open a Search Engine that allows you to locate each instance of a keyword across all areas of the GMOMMWEB program. The Search will list all instances found. Click the link to go to that particular page of GMOMMWEB. Open a Movie List File in Adobe Reader to browse or search for a particular QuickTime or Flash Movie. Each Linked Movie will open in the appropriate movie player when the movie link is clicked.
Embed this Program Add this Program to your website by copying the code below. Preview Preview.
Gray Matter Download For Mac Download
Talairach Client - Version 2.4.3 The Talairach Client reports Talairach labels for user-defined coordinates. Coordinate data can be manually input through the interface or read from a file. Input data files should be tab-, space-, or comma-delimited and arranged in x,y,z order. There are options to search for the single point, search range, or nearest gray matter.
The results are shown on the interface or written to a file that can be viewed in a text editor or imported into a spreadsheet. For a more thorough description, read the.
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1 VU Medical Center Alzheimer Center, Amsterdam, The Netherlands. Electronic address: [email protected]. 2 Department of Radiology and Nuclear Medicine, VU Medical Center, Amsterdam, The Netherlands. 3 VU Medical Center Alzheimer Center, Amsterdam, The Netherlands; Department of Radiology and Nuclear Medicine, VU Medical Center, Amsterdam, The Netherlands.
4 VU Medical Center Alzheimer Center, Amsterdam, The Netherlands; Department of Radiology and Nuclear Medicine, VU Medical Center, Amsterdam, The Netherlands; Department of Diagnostic and Interventional Neuroradiology, Hannover Medical School, Hannover, Germany. 5 VU Medical Center Alzheimer Center, Amsterdam, The Netherlands. We aimed to study if patients with dementia with Lewy bodies (DLB) who have concomitant Alzheimer's disease (AD) pathology (detected antemortem by cerebrospinal fluid CSF biomarkers) have additional loss of gray matter volume. Ninety-eight DLB patients were divided into a 'pure DLB' (DLB/AD-, n = 62) and a 'mixed DLB' group (DLB/AD+, n = 36) and matched for age and symptom duration to 84 AD patients and 75 controls.
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We compared visual atrophy ratings, and in a subset, we analyzed cortical thickness and subcortical gray matter volumes. DLB/AD+ patients had more pronounced medial temporal lobe atrophy (MTA) compared to DLB/AD- (mean total MTA score 2.5 vs. 1.3, p = 0.02). Global and parietal atrophy scores were comparable between the 3 dementia groups and differed from controls. MTA score was associated with CSF Aβ-42, while posterior cortical and global atrophy scores were associated with CSF tau. Cortical thinning was found in DLB/AD-, DLB/AD+, and AD compared to controls. Concomitant AD pathology seems to cause additional (hippocampal) atrophy in DLB, and this may contribute to a more devastating disease course in DLB/AD+ patients.